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Experimental and human metabolic literature demonstrates that GH promotes fatty acid mobilization, shifts substrate preference toward fat oxidation, and alters carbohydrate-lipid partitioning during fasting and metabolic stress states, making GH secretagogue systems useful tools for investigating adipose tissue signaling, fuel selection, and the endocrine regulation of energy metabolism [8]

Additionally, TMAO dose-dependently suppresses TRPV4-mediated Ca 2+ influx, blocking the TRPV4-NO pathway to reduce endothelial NO production and compromise vasodilation (Zhang et al., 2024)

Children, the elderly and people with chronic illnesses are the most vulnerable